Why All Anti-Aging Products Only Treat the Signs of Aging
To date, all anti-aging products have been aimed at correcting skin damage and treating aged skin after it has already occurred.
There are an endless number of creams that claim to restore the elastin in your skin and boost the collagen to mask those lines and wrinkles.
These days, Botox is the most common cosmetic operation, which when injected into the skin causes muscle paralysis and thus flattening and softening of facial creases of the skin.
This multi-billion dollar beauty and cosmetic industry relies on the vanity and inherent need of our population to want to look better and, more importantly, look younger.
Skin care is a big business.
And although there is nothing wrong with that, the truth of the matter is that the skin care and anti-aging industry relies on consumers consistently returning to purchase their creams and to use their latest and greatest products.
Most dermatological research currently focuses on treating skin damage and correcting aged skin after it has already occurred, and this has precisely been the focus of the skin care industry.
Until now.
A breakthrough discovery, verified by Purdue University, has revealed that there is an enzyme within the cell membrane of the skin epidermis which generates free radicals increasingly with age, causing the skin to age quicker as we get older.
This enzyme, termed age-related NADH oxidase (or arNOX), is an internal source of free radicals within the skin.
These free radicals, which can also be generated from exposure to the environment such as from the sun or cigarette smoke, cause damage to collagen and elastin proteins in the skin, resulting in a loss of firmness and noticeably more lines and wrinkles.
In fact, clinical studies conducted at Stanford University have shown that those who have higher serum levels of arNOX look on average seven years older.
This recent discovery has allowed the dermatological field to gain a deeper insight into the process of skin aging, and gives scientists the ability to research fundamental mechanisms that may combine with the known external sources which lead to an accelerated aging process.
There are an endless number of creams that claim to restore the elastin in your skin and boost the collagen to mask those lines and wrinkles.
These days, Botox is the most common cosmetic operation, which when injected into the skin causes muscle paralysis and thus flattening and softening of facial creases of the skin.
This multi-billion dollar beauty and cosmetic industry relies on the vanity and inherent need of our population to want to look better and, more importantly, look younger.
Skin care is a big business.
And although there is nothing wrong with that, the truth of the matter is that the skin care and anti-aging industry relies on consumers consistently returning to purchase their creams and to use their latest and greatest products.
Most dermatological research currently focuses on treating skin damage and correcting aged skin after it has already occurred, and this has precisely been the focus of the skin care industry.
Until now.
A breakthrough discovery, verified by Purdue University, has revealed that there is an enzyme within the cell membrane of the skin epidermis which generates free radicals increasingly with age, causing the skin to age quicker as we get older.
This enzyme, termed age-related NADH oxidase (or arNOX), is an internal source of free radicals within the skin.
These free radicals, which can also be generated from exposure to the environment such as from the sun or cigarette smoke, cause damage to collagen and elastin proteins in the skin, resulting in a loss of firmness and noticeably more lines and wrinkles.
In fact, clinical studies conducted at Stanford University have shown that those who have higher serum levels of arNOX look on average seven years older.
This recent discovery has allowed the dermatological field to gain a deeper insight into the process of skin aging, and gives scientists the ability to research fundamental mechanisms that may combine with the known external sources which lead to an accelerated aging process.
